this page in germandeutsche Version hier
You find this site in SSL (https://) as https://4ju.org/ or http://fight-cancer.4uj.org or http://fight-cancer.4ju.org
Here just some
nutritional advice that may give you support in your fight against cancer:
- avoid sugar (as completely as you can, also carbohydrates, (products made of) white flour ... sugar is everywhere in 'ready made meal' or in spice mixtures, even in stevia products!)
- Good curry-mixtures are pure medicine. Pepper/chili and oil boost the power of turmeric powder.
- All sorts of gym: cycling, walking, running, gym, in-house-gym, jogging
- Aspirin in very low dosage (100mg and less)
- Broccoli-seed, broccoli and cauliflower. But all sorts of cabbage have a positive effect
- Virgin coconut oil !!! (no chemically treated/hardened coconut oil, which is harmfull)
- Green tea. 4 cups a day
- No artificial vitamins. But all sorts of legumes or fruits
- Humulus and resveratrol together with quercetin(apples,onions,garlic)
- Omega3 (fish oil) (higher dosage necessary algae, seaweeds).
- Olive oil , linseed oil , cocos-oil, avocados
- Nuts (nearly all sorts of nuts)
- Linseed
- Chili , in combination with turmeric powder, ginger
- Garlic, onions
- Shitake (not sure)
- Tea tree oil, virgin mint oil (not for ingestion)
- no fat, except cocos-oil (no animal fat)
- Seeds (lot of experience necessary)
- No alcohol (degrades your immune system and you should do everything for a strong immune system)
Growing link between antibiotics and colon cancer
First evidence: Parkinson's and Alzheimer's disease caused by fungi
Enhancement of melanoma aggressiveness via p38-MAPK, HIF-1α pathways, and metabolic reprogramming induced by Candida albicans
(Scientific Reports, 2026;
doi: 10.1038/s41598-025-24055-y) Quelle: Scientific Reports, University of the Basque Country Recent studies have increasingly focused on the role of fungi,
including Candida albicans, in carcinogenesis. Since C.
albicans is a component of the human microbiota, particularly on the
skin, we investigated its effect on the phenotype and signalling
pathways of melanoma cells. Assays for migration, adhesion,
angiogenesis, and hepatic metastasis showed that C. albicans
promotes a more malignant phenotype in melanoma cells. At the
transcriptomic level, C. albicans increased the expression of
VEGF (Vegfa), and genes associated with MAPK and HIF-1 signalling
pathways, and with aerobic glycolysis. Further in vitro analysis
revealed that TLRs and EphA2 receptors are involved in the recognition
of live C. albicans, stimulating VEGF secretion and expression of
the AP-1 transcription factor component c-Fos through p38-MAPK and
HIF-1α. These pathways also regulate the expression of other AP-1
constituents such as Atf3, Jun, and Jund. Moreover,
p38-MAPK regulates glycolytic genes like Hk2, Slc2a1, and
Eno2. In conclusion, C. albicans activates the p38-MAPK/c-Fos/AP-1
and HIF-1/HIF-1α/c-Fos/AP-1 pathways in melanoma cells, promoting a pro-angiogenic
environment and metabolic reprogramming. Therefore, this study clarifies
the impact of C. albicans on melanoma cells, which can lead to
the use of antifungal therapies as complementary to traditional
treatments for melanoma.
Abstract
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